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Interleukin 35 protects cardiomyocytes following ischemia/reperfusion-induced apoptosis via activation of mitochondrial STAT3.
Zhou, Fengyun; Feng, Ting; Lu, Xiangqi; Wang, Huicheng; Chen, Yangping; Zhang, Qiuxia; Zhang, Xinlu; Xiu, Jiancheng.
Afiliação
  • Zhou F; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Feng T; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Lu X; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Wang H; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Chen Y; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Zhang Q; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Zhang X; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
  • Xiu J; Department of Cardiology, State Key Laboratory of Organ Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Acta Biochim Biophys Sin (Shanghai) ; 53(4): 410-418, 2021 Mar 26.
Article em En | MEDLINE | ID: mdl-33619515
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Interleucinas / Apoptose / Proteínas Mitocondriais / Miócitos Cardíacos / Fator de Transcrição STAT3 / Mitocôndrias Cardíacas Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2021 Tipo de documento: Article
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Interleucinas / Apoptose / Proteínas Mitocondriais / Miócitos Cardíacos / Fator de Transcrição STAT3 / Mitocôndrias Cardíacas Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2021 Tipo de documento: Article