Pathogenesis of Borrelia burgdorferi and Babesia microti in TLR4-Competent and TLR4-dysfunctional C3H mice.

Akoolo, Lavoisier; Djokic, Vitomir; Rocha, Sandra C; Parveen, Nikhat

Akoolo, Lavoisier; Djokic, Vitomir; Rocha, Sandra C; Parveen, Nikhat.

Afiliação

Akoolo L; Department of Microbiology, Biochemistry and Molecular Genetics, Rutgers New Jersey Medical School, Newark, New Jersey, USA.
Djokic V; Department of Microbiology, Biochemistry and Molecular Genetics, Rutgers New Jersey Medical School, Newark, New Jersey, USA.
Rocha SC; Department of Microbiology, Biochemistry and Molecular Genetics, Rutgers New Jersey Medical School, Newark, New Jersey, USA.
Parveen N; Department of Microbiology, Biochemistry and Molecular Genetics, Rutgers New Jersey Medical School, Newark, New Jersey, USA.

Cell Microbiol ; 23(9): e13350, 2021 09.

Article em En | MEDLINE | ID: mdl-33938125

Assuntos

Babesia microti; Babesiose; Borrelia burgdorferi; Doença de Lyme; Animais; Células HEK293; Humanos; Camundongos; Camundongos Endogâmicos C3H; Receptor 4 Toll-Like/genética

Palavras-chave

Babesia microti; Babesiosis; Borrelia burgdorferi; Lyme disease; co-infection; inflammation; toll like receptor 4

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Babesiose / Doença de Lyme / Borrelia burgdorferi / Babesia microti Tipo de estudo: Etiology_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2021 Tipo de documento: Article

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