Loss of CC2D1A in Glutamatergic Neurons Results in Autistic-Like Features in Mice.

Yang, Cheng-Yi; Hung, Yu-Chieh; Cheng, Kuan-Hsiang; Ling, Pin; Hsu, Kuei-Sen

Yang, Cheng-Yi; Hung, Yu-Chieh; Cheng, Kuan-Hsiang; Ling, Pin; Hsu, Kuei-Sen.

Afiliação

Yang CY; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, 70101, Taiwan.
Hung YC; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, 70101, Taiwan.
Cheng KH; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, 70101, Taiwan.
Ling P; Department of Microbiology & Immunology, College of Medicine, National Cheng Kung University, Tainan, 70101, Taiwan.
Hsu KS; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, 70101, Taiwan. richard@mail.ncku.edu.tw.

Neurotherapeutics ; 18(3): 2021-2039, 2021 07.

Article em En | MEDLINE | ID: mdl-34132974

Assuntos

Transtorno Autístico/metabolismo; Ácido Glutâmico/metabolismo; Neurônios/metabolismo; Proteínas Repressoras/deficiência; Interação Social; Animais; Animais Recém-Nascidos; Transtorno Autístico/genética; Masculino; Camundongos; Camundongos Endogâmicos C57BL; Camundongos Knockout; Técnicas de Cultura de Órgãos; Proteínas Repressoras/genética

Palavras-chave

Autism spectrum disorder; CC2D1A; Excitation/inhibition balance; Medial prefrontal cortex; Minocycline

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas Repressoras / Transtorno Autístico / Ácido Glutâmico / Interação Social / Neurônios Limite: Animals Idioma: En Ano de publicação: 2021 Tipo de documento: Article

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