Ubiquitination and receptor-mediated mitophagy converge to eliminate oxidation-damaged mitochondria during hypoxia.

Sulkshane, Prasad; Ram, Jonathan; Thakur, Anita; Reis, Noa; Kleifeld, Oded; Glickman, Michael H

Sulkshane, Prasad; Ram, Jonathan; Thakur, Anita; Reis, Noa; Kleifeld, Oded; Glickman, Michael H.

Afiliação

Sulkshane P; The Faculty of Biology, Technion Israel Institute of Technology, Haifa, 32000, Israel.
Ram J; The Faculty of Biology, Technion Israel Institute of Technology, Haifa, 32000, Israel.
Thakur A; The Faculty of Biology, Technion Israel Institute of Technology, Haifa, 32000, Israel.
Reis N; The Faculty of Biology, Technion Israel Institute of Technology, Haifa, 32000, Israel.
Kleifeld O; The Faculty of Biology, Technion Israel Institute of Technology, Haifa, 32000, Israel.
Glickman MH; The Faculty of Biology, Technion Israel Institute of Technology, Haifa, 32000, Israel. Electronic address: glickman@technion.ac.il.

Redox Biol ; 45: 102047, 2021 09.

Article em En | MEDLINE | ID: mdl-34175667

Assuntos

Mitocôndrias; Mitofagia; Células HeLa; Humanos; Hipóxia/metabolismo; Mitocôndrias/metabolismo; Estresse Oxidativo; Ubiquitinação

Palavras-chave

HIF-1α; Hypoxia; Mitochondria; Mitophagy; Oxidative stress; Proteasome; Ubiquitin

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Mitofagia / Mitocôndrias Limite: Humans Idioma: En Ano de publicação: 2021 Tipo de documento: Article

Texto completo

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PubMed Links

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Mitofagia / Mitocôndrias Limite: Humans Idioma: En Ano de publicação: 2021 Tipo de documento: Article