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Combined glyoxalase 1 dysfunction and vitamin B6 deficiency in a schizophrenia model system causes mitochondrial dysfunction in the prefrontal cortex.
Toriumi, Kazuya; Berto, Stefano; Koike, Shin; Usui, Noriyoshi; Dan, Takashi; Suzuki, Kazuhiro; Miyashita, Mitsuhiro; Horiuchi, Yasue; Yoshikawa, Akane; Asakura, Mai; Nagahama, Kenichiro; Lin, Hsiao-Chun; Sugaya, Yuki; Watanabe, Takaki; Kano, Masanobu; Ogasawara, Yuki; Miyata, Toshio; Itokawa, Masanari; Konopka, Genevieve; Arai, Makoto.
Afiliação
  • Toriumi K; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan; Department of Neuroscience, University of Texas Southwestern Medical Center, Dallas, TX 75390-9111, USA.
  • Berto S; Department of Neuroscience, University of Texas Southwestern Medical Center, Dallas, TX 75390-9111, USA; Department of Neuroscience, Medical University of South Carolina, Charleston, SC 29403, USA.
  • Koike S; Department of Analytical Biochemistry, Meiji Pharmaceutical University, Tokyo 204-8588, Japan.
  • Usui N; Department of Neuroscience, University of Texas Southwestern Medical Center, Dallas, TX 75390-9111, USA; Center for Medical Research and Education, Graduate School of Medicine, Osaka University, Osaka, 565-0871, Japan; Department of Neuroscience and Cell Biology, Graduate School of Medicine, Osaka U
  • Dan T; Division of Molecular Medicine and Therapy, Tohoku University Graduate School of Medicine, Sendai, 980-8575, Japan.
  • Suzuki K; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan; Department of Psychiatry, Graduate School of Medicine, Shinshu University, Nagano, 390-8621, Japan.
  • Miyashita M; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan.
  • Horiuchi Y; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan.
  • Yoshikawa A; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan; Department of Psychiatry and Behavioral Science, Graduate School of Medicine, Juntendo University, Tokyo, 113-8421, Japan.
  • Asakura M; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan.
  • Nagahama K; Department of Neurophysiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan.
  • Lin HC; Department of Neurophysiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan.
  • Sugaya Y; Department of Neurophysiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan.
  • Watanabe T; Department of Neurophysiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan.
  • Kano M; Department of Neurophysiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan.
  • Ogasawara Y; Department of Analytical Biochemistry, Meiji Pharmaceutical University, Tokyo 204-8588, Japan.
  • Miyata T; Division of Molecular Medicine and Therapy, Tohoku University Graduate School of Medicine, Sendai, 980-8575, Japan.
  • Itokawa M; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan.
  • Konopka G; Department of Neuroscience, University of Texas Southwestern Medical Center, Dallas, TX 75390-9111, USA.
  • Arai M; Schizophrenia Research Project, Department of Psychiatry and Behavioral Sciences, Tokyo Metropolitan Institute of Medical Science, Tokyo, 156-8506, Japan. Electronic address: arai-mk@igakuken.or.jp.
Redox Biol ; 45: 102057, 2021 09.
Article em En | MEDLINE | ID: mdl-34198071
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Deficiência de Vitamina B 6 / Esquizofrenia / Lactoilglutationa Liase Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2021 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Deficiência de Vitamina B 6 / Esquizofrenia / Lactoilglutationa Liase Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2021 Tipo de documento: Article