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MicroRNA (miR)-590-3p alleviates high-glucose induced renal tubular epithelial cell damage by targeting C-X3-C motif chemokine ligand 1 (CX3CL1) in diabetic nephropathy.
Yun, Jie; Ren, Jinyu; Liu, Yufei; Dai, Lijuan; Song, Liqun; Ma, Xiaopeng; Luo, Shan; Song, Yexu.
Afiliação
  • Yun J; Department of Nephrology, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Ren J; Department of Encephalopathy, Second Hospital Affiliated to Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Liu Y; Department of Blood Purification, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Dai L; Department of Nephrology, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Song L; Department of Nephrology, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Ma X; Department of Nephrology, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Luo S; Department of Nephrology, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
  • Song Y; Department of Science and Technology, Heilongjiang University of Chinese Medicine, Heilongjiang, China.
Bioengineered ; 13(1): 634-644, 2022 01.
Article em En | MEDLINE | ID: mdl-34898373
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: MicroRNAs / Nefropatias Diabéticas / Células Epiteliais / Quimiocina CX3CL1 / Glucose / Túbulos Renais Tipo de estudo: Prognostic_studies Limite: Adult / Aged / Female / Humans / Male / Middle aged Idioma: En Ano de publicação: 2022 Tipo de documento: Article
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: MicroRNAs / Nefropatias Diabéticas / Células Epiteliais / Quimiocina CX3CL1 / Glucose / Túbulos Renais Tipo de estudo: Prognostic_studies Limite: Adult / Aged / Female / Humans / Male / Middle aged Idioma: En Ano de publicação: 2022 Tipo de documento: Article