Your browser doesn't support javascript.
loading
Amyloid-like aggregating proteins cause lysosomal defects in neurons via gain-of-function toxicity.
Riera-Tur, Irene; Schäfer, Tillman; Hornburg, Daniel; Mishra, Archana; da Silva Padilha, Miguel; Fernández-Mosquera, Lorena; Feigenbutz, Dennis; Auer, Patrick; Mann, Matthias; Baumeister, Wolfgang; Klein, Rüdiger; Meissner, Felix; Raimundo, Nuno; Fernández-Busnadiego, Rubén; Dudanova, Irina.
Afiliação
  • Riera-Tur I; Department of Molecules-Signaling-Development, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Schäfer T; Molecular Neurodegeneration Group, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Hornburg D; Department of Molecular Structural Biology, Max Planck Institute of Biochemistry, Martinsried, Germany.
  • Mishra A; Department of Proteomics and Signal Transduction, Max Planck Institute of Biochemistry, Martinsried, Germany.
  • da Silva Padilha M; Experimental Systems Immunology Group, Max Planck Institute of Biochemistry, Martinsried, Germany.
  • Fernández-Mosquera L; Department of Molecules-Signaling-Development, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Feigenbutz D; Department of Molecules-Signaling-Development, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Auer P; Molecular Neurodegeneration Group, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Mann M; The William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, UK.
  • Baumeister W; Department of Molecules-Signaling-Development, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Klein R; Molecular Neurodegeneration Group, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Meissner F; Department of Molecules-Signaling-Development, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Raimundo N; Molecular Neurodegeneration Group, Max Planck Institute of Neurobiology, Martinsried, Germany.
  • Fernández-Busnadiego R; Department of Proteomics and Signal Transduction, Max Planck Institute of Biochemistry, Martinsried, Germany.
  • Dudanova I; Department of Molecular Structural Biology, Max Planck Institute of Biochemistry, Martinsried, Germany.
Life Sci Alliance ; 5(3)2022 03.
Article em En | MEDLINE | ID: mdl-34933920

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas Amiloidogênicas / Mutação com Ganho de Função / Neurônios Tipo de estudo: Etiology_studies Idioma: En Ano de publicação: 2022 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas Amiloidogênicas / Mutação com Ganho de Função / Neurônios Tipo de estudo: Etiology_studies Idioma: En Ano de publicação: 2022 Tipo de documento: Article