Endoplasmic Reticulum Stress/Ca<sup>2+</sup>-Calmodulin-Dependent Protein Kinase/Signal Transducer and Activator of Transcription 3 Pathway Plays a Role in the Regulation of Cellular Zinc Deficiency in Myocardial Ischemia/Reperfusion Injury.

Zhao, Huanhuan; Liu, Dan; Yan, Qiumei; Bian, Xiyun; Yu, Jing; Wang, Jingjing; Cheng, Xinxin; Xu, Zhelong

Endoplasmic Reticulum Stress/Ca²⁺-Calmodulin-Dependent Protein Kinase/Signal Transducer and Activator of Transcription 3 Pathway Plays a Role in the Regulation of Cellular Zinc Deficiency in Myocardial Ischemia/Reperfusion Injury.

Zhao, Huanhuan; Liu, Dan; Yan, Qiumei; Bian, Xiyun; Yu, Jing; Wang, Jingjing; Cheng, Xinxin; Xu, Zhelong.

Afiliação

Zhao H; Department of Physiology and Pathophysiology, Tianjin Medical University, Tianjin, China.
Liu D; Department of Physiology and Pathophysiology, Tianjin Medical University, Tianjin, China.
Yan Q; Department of Physiology and Pathophysiology, Tianjin Medical University, Tianjin, China.
Bian X; Central Laboratory, Tianjin, China.
Yu J; Tianjin Key Laboratory of Epigenetics for Organ Development in Preterm Infants, The Fifth Central Hospital of Tianjin, Tianjin, China.
Wang J; Department of Physiology and Pathophysiology, Tianjin Medical University, Tianjin, China.
Cheng X; Department of Physiology and Pathophysiology, Tianjin Medical University, Tianjin, China.
Xu Z; Department of Physiology and Pathophysiology, Tianjin Medical University, Tianjin, China.

Front Physiol ; 12: 736920, 2021.

Article em En | MEDLINE | ID: mdl-35069232