Your browser doesn't support javascript.
loading
Activation of CD44/PAK1/AKT signaling promotes resistance to FGFR1 inhibition in squamous-cell lung cancer.
Elakad, Omar; Häupl, Björn; Labitzky, Vera; Yao, Sha; Küffer, Stefan; von Hammerstein-Equord, Alexander; Danner, Bernhard C; Jücker, Manfred; Urlaub, Henning; Lange, Tobias; Ströbel, Philipp; Oellerich, Thomas; Bohnenberger, Hanibal.
Afiliação
  • Elakad O; Institute of Pathology, University Medical Center, Goettingen, Germany.
  • Häupl B; Department of Medicine II, Hematology/Oncology, Goethe University, Frankfurt, Germany.
  • Labitzky V; German Cancer Research Center and German Cancer Consortium, Heidelberg, Germany.
  • Yao S; Frankfurt Cancer Institute, Goethe University, Frankfurt, Germany.
  • Küffer S; Institute for Anatomy and Experimental Morphology, University Cancer Center, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • von Hammerstein-Equord A; Institute of Pathology, University Medical Center, Goettingen, Germany.
  • Danner BC; Institute of Pathology, University Medical Center, Goettingen, Germany.
  • Jücker M; Department of Thoracic and Cardiovascular Surgery, University Medical Center, Goettingen, Germany.
  • Urlaub H; Department of Thoracic and Cardiovascular Surgery, University Medical Center, Goettingen, Germany.
  • Lange T; Center for Experimental Medicine, Institute of Biochemistry and Signal Transduction, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Ströbel P; Bioanalytical Mass Spectrometry Group, Max Planck Institute for Biophysical Chemistry, Goettingen, Germany.
  • Oellerich T; Institute for Anatomy and Experimental Morphology, University Cancer Center, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Bohnenberger H; Institute of Pathology, University Medical Center, Goettingen, Germany.
NPJ Precis Oncol ; 6(1): 52, 2022 Jul 19.
Article em En | MEDLINE | ID: mdl-35853934

Texto completo: 1 Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Ano de publicação: 2022 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Ano de publicação: 2022 Tipo de documento: Article