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ST3GAL5-catalyzed gangliosides inhibit TGF-ß-induced epithelial-mesenchymal transition via TßRI degradation.
Zhang, Jing; van der Zon, Gerard; Ma, Jin; Mei, Hailiang; Cabukusta, Birol; Agaser, Cedrick C; Madunic, Katarina; Wuhrer, Manfred; Zhang, Tao; Ten Dijke, Peter.
Afiliação
  • Zhang J; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, The Netherlands.
  • van der Zon G; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, The Netherlands.
  • Ma J; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, The Netherlands.
  • Mei H; Sequencing Analysis Support Core, Leiden University Medical Center, Leiden, The Netherlands.
  • Cabukusta B; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, The Netherlands.
  • Agaser CC; Sequencing Analysis Support Core, Leiden University Medical Center, Leiden, The Netherlands.
  • Madunic K; Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.
  • Wuhrer M; Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.
  • Zhang T; Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.
  • Ten Dijke P; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, The Netherlands.
EMBO J ; 42(2): e110553, 2023 01 16.
Article em En | MEDLINE | ID: mdl-36504224

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Fator de Crescimento Transformador beta / Neoplasias Pulmonares Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2023 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Fator de Crescimento Transformador beta / Neoplasias Pulmonares Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2023 Tipo de documento: Article