Copper Exposure Induces Epithelial-Mesenchymal Transition-Related Fibrotic Change via Autophagy and Increase Risk of Lung Fibrosis in Human.

Chiou, Hsin-Ying Clair; Wang, Chih-Wen; Chen, Szu-Chia; Tsai, Mei-Lan; Lin, Ming-Hong; Hung, Chih-Hsing; Kuo, Chao-Hung

Chiou, Hsin-Ying Clair; Wang, Chih-Wen; Chen, Szu-Chia; Tsai, Mei-Lan; Lin, Ming-Hong; Hung, Chih-Hsing; Kuo, Chao-Hung.

Afiliação

Chiou HC; Teaching and Research Center, Kaohsiung Municipal Siaogang Hospital, Kaohsiung Medical University, Kaohsiung 807, Taiwan.
Wang CW; Kaohsiung Medical University Hospital, Kaohsiung Medical University, Kaohsiung 807, Taiwan.
Chen SC; Department of Applied Chemistry, National Chi Nan University, Nantou 540, Taiwan.
Tsai ML; Division of Hepatobiliary, Department of Internal Medicine, Kaohsiung Medical University Hospital, Kaohsiung Medical University, Kaohsiung 807, Taiwan.
Lin MH; Department of Internal Medicine, Kaohsiung Municipal Siaogang Hospital, Kaohsiung Medical University, Kaohsiung 807, Taiwan.
Hung CH; Research Center for Environmental Medicine, Kaohsiung Medical University, Kaohsiung 807, Taiwan.
Kuo CH; Department of Internal Medicine, Kaohsiung Municipal Siaogang Hospital, Kaohsiung Medical University, Kaohsiung 807, Taiwan.

Antioxidants (Basel) ; 12(2)2023 Feb 20.

Article em En | MEDLINE | ID: mdl-36830091

Palavras-chave

autophagy; copper; epithelial-mesenchymal transition; flavonoid; pulmonary fibrosis

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Texto completo: 1 Base de dados: MEDLINE Tipo de estudo: Etiology_studies / Prognostic_studies / Risk_factors_studies Idioma: En Ano de publicação: 2023 Tipo de documento: Article

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