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MAT1A Suppression by the CTBP1/HDAC1/HDAC2 Transcriptional Complex Induces Immune Escape and Reduces Ferroptosis in Hepatocellular Carcinoma.
Li, Yaqin; Hu, Guoxin; Huang, Furong; Chen, Mingtai; Chen, Yihua; Xu, Youhua; Tong, Guangdong.
Afiliação
  • Li Y; Faculty of Chinese Medicine, Macau University of Science and Technology, Taipa, Macao, China; Department of Infectious Disease, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
  • Hu G; Department of Infectious Disease, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
  • Huang F; Department of Liver Disease, Shenzhen Traditional Chinese Medicine Hospital, the Fourth Clinical Medical College of Guangzhou University of Chinese Medicine, Shenzhen, Guangdong, China.
  • Chen M; Department of Cardiovascular Disease, Shenzhen Traditional Chinese Medicine Hospital, the Fourth Clinical Medical College of Guangzhou University of Chinese Medicine, Shenzhen, Guangdong, China.
  • Chen Y; Department of Infectious Disease, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
  • Xu Y; Faculty of Chinese Medicine, Macau University of Science and Technology, Taipa, Macao, China. Electronic address: yhxu@must.edu.mo.
  • Tong G; Faculty of Chinese Medicine, Macau University of Science and Technology, Taipa, Macao, China; Department of Liver Disease, Shenzhen Traditional Chinese Medicine Hospital, the Fourth Clinical Medical College of Guangzhou University of Chinese Medicine, Shenzhen, Guangdong, China. Electronic address:
Lab Invest ; 103(8): 100180, 2023 08.
Article em En | MEDLINE | ID: mdl-37230466

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Carcinoma Hepatocelular / Ferroptose / Neoplasias Hepáticas Tipo de estudo: Risk_factors_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2023 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Carcinoma Hepatocelular / Ferroptose / Neoplasias Hepáticas Tipo de estudo: Risk_factors_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2023 Tipo de documento: Article