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ER stress increases expression of intracellular calcium channel RyR1 to modify Ca2+ homeostasis in pancreatic beta cells.
Zhang, Irina X; Herrmann, Andrea; Leon, Juan; Jeyarajan, Sivakumar; Arunagiri, Anoop; Arvan, Peter; Gilon, Patrick; Satin, Leslie S.
Afiliação
  • Zhang IX; Department of Pharmacology and Brehm Diabetes Research Center, University of Michigan Medical School, Ann Arbor, Michigan, USA.
  • Herrmann A; Department of Pharmacology and Brehm Diabetes Research Center, University of Michigan Medical School, Ann Arbor, Michigan, USA.
  • Leon J; J. Crayton Pruitt Family Department of Biomedical Engineering, University of Florida, Gainesville, Florida, USA.
  • Jeyarajan S; Department of Pharmacology and Brehm Diabetes Research Center, University of Michigan Medical School, Ann Arbor, Michigan, USA.
  • Arunagiri A; Department of Metabolism, Endocrinology & Diabetes, University of Michigan, Ann Arbor, Michigan, USA.
  • Arvan P; Department of Metabolism, Endocrinology & Diabetes, University of Michigan, Ann Arbor, Michigan, USA.
  • Gilon P; Pole of Endocrinology, Diabetes and Nutrition (EDIN), Institute of Experimental and Clinical Research (IREC), Université Catholique de Louvain, Brussels, Belgium.
  • Satin LS; Department of Pharmacology and Brehm Diabetes Research Center, University of Michigan Medical School, Ann Arbor, Michigan, USA. Electronic address: lsatin@umich.edu.
J Biol Chem ; 299(8): 105065, 2023 08.
Article em En | MEDLINE | ID: mdl-37468098
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Canal de Liberação de Cálcio do Receptor de Rianodina / Sinalização do Cálcio / Células Secretoras de Insulina / Estresse do Retículo Endoplasmático Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article
Texto completo
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Canal de Liberação de Cálcio do Receptor de Rianodina / Sinalização do Cálcio / Células Secretoras de Insulina / Estresse do Retículo Endoplasmático Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article