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METTL3-mediated N6-methyladenosine exacerbates ferroptosis via m6A-IGF2BP2-dependent mitochondrial metabolic reprogramming in sepsis-induced acute lung injury.
Zhang, Hao; Wu, Dan; Wang, Yanghanzhao; Guo, Kefang; Spencer, Charles B; Ortoga, Lilibeth; Qu, Mengdi; Shi, Yuxin; Shao, Yuwen; Wang, Zhiping; Cata, Juan P; Miao, Changhong.
Afiliação
  • Zhang H; Department of Anesthesiology, Zhongshan Hospital, Fudan University, Shanghai, China.
  • Wu D; Shanghai Key Laboratory of Perioperative Stress and Protection, Shanghai, China.
  • Wang Y; Department of Anesthesiology, Shanghai Medical College, Fudan University, Shanghai, China.
  • Guo K; Department of Anesthesiology, Zhongshan Hospital, Fudan University, Shanghai, China.
  • Spencer CB; Shanghai Key Laboratory of Perioperative Stress and Protection, Shanghai, China.
  • Ortoga L; Department of Anesthesiology, Shanghai Medical College, Fudan University, Shanghai, China.
  • Qu M; Department of Anesthesiology, Zhongshan Hospital, Fudan University, Shanghai, China.
  • Shi Y; Shanghai Key Laboratory of Perioperative Stress and Protection, Shanghai, China.
  • Shao Y; Department of Anesthesiology, Shanghai Medical College, Fudan University, Shanghai, China.
  • Wang Z; Department of Anesthesiology, Zhongshan Hospital, Fudan University, Shanghai, China.
  • Cata JP; Shanghai Key Laboratory of Perioperative Stress and Protection, Shanghai, China.
  • Miao C; Department of Anesthesiology, Shanghai Medical College, Fudan University, Shanghai, China.
Clin Transl Med ; 13(9): e1389, 2023 09.
Article em En | MEDLINE | ID: mdl-37715457
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Sepse / Lesão Pulmonar Aguda / Ferroptose Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Sepse / Lesão Pulmonar Aguda / Ferroptose Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article