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Src inhibition modulates AMBRA1-mediated mitophagy to counteract endothelial-to-mesenchymal transition in renal allograft fibrosis.
Gui, Zeping; Liu, Xuzhong; Xu, Zhen; Feng, Dengyuan; Hang, Zhou; Zheng, Ming; Chen, Hao; Fei, Shuang; Sun, Li; Tao, Jun; Han, Zhijian; Ju, Xiaobin; Gu, Min; Tan, Ruoyun; Wang, Zijie.
Afiliação
  • Gui Z; Department of Urology, The Second Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Liu X; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Xu Z; Department of Urology, Huai'an First People's Hospital, Nanjing Medical University, Huai'an, China.
  • Feng D; Department of Urology, Affiliated Clinical College of Xuzhou Medical University, Huai'an, China.
  • Hang Z; Department of Urology, The Affiliated Taizhou People's Hospital of Nanjing Medical University, Taizhou, China.
  • Zheng M; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Chen H; Department of Urology, The Second Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Fei S; Department of Urology, The Second Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Sun L; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Tao J; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Han Z; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Ju X; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Gu M; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Tan R; Department of Urology, The First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
  • Wang Z; Department of Urology, The Second Affiliated Hospital with Nanjing Medical University, Nanjing, China.
Cell Prolif ; : e13699, 2024 Jun 29.
Article em En | MEDLINE | ID: mdl-38943534

Texto completo: 1 Base de dados: MEDLINE Idioma: En Ano de publicação: 2024 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Idioma: En Ano de publicação: 2024 Tipo de documento: Article