Spontaneous NETosis and type I IFN signaling activation in resting neutrophils of chronic granulomatous disease patients with <i>CYBB</i> mutations.

Spontaneous NETosis and type I IFN signaling activation in resting neutrophils of chronic granulomatous disease patients with CYBB mutations.

Lu, Zhengjing; Mou, Wenjun; He, Jianxin; Ge, Xuedong; Wang, Xiaolin; Gui, Jingang.

Afiliação

Lu Z; Laboratory of Tumor Immunology, Key Laboratory of Major Diseases in Children, Ministry of Education, Beijing Pediatric Research Institute, Beijing Children's Hospital, Capital Medical University, National Center for Children's Health, Beijing 100045, China.
Mou W; Laboratory of Tumor Immunology, Key Laboratory of Major Diseases in Children, Ministry of Education, Beijing Pediatric Research Institute, Beijing Children's Hospital, Capital Medical University, National Center for Children's Health, Beijing 100045, China.
He J; Department of Respiratory Medicine, Beijing Children's Hospital, Capital Medical University, National Center for Children's Health, China National Clinical Research Center for Respiratory Diseases, Beijing 100045, China.
Ge X; Information Center, PLA Rocket Force Characteristic Medical Center, Beijing 100088, China.
Wang X; Laboratory of Tumor Immunology, Key Laboratory of Major Diseases in Children, Ministry of Education, Beijing Pediatric Research Institute, Beijing Children's Hospital, Capital Medical University, National Center for Children's Health, Beijing 100045, China.
Gui J; Laboratory of Tumor Immunology, Key Laboratory of Major Diseases in Children, Ministry of Education, Beijing Pediatric Research Institute, Beijing Children's Hospital, Capital Medical University, National Center for Children's Health, Beijing 100045, China.

Genes Dis ; 11(6): 101118, 2024 Nov.

Article em En | MEDLINE | ID: mdl-39070548

Texto completo: 1 Base de dados: MEDLINE Idioma: En Ano de publicação: 2024 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Idioma: En Ano de publicação: 2024 Tipo de documento: Article