Detalhe da pesquisa
1.
Uninephrectomy and class II PI3K-C2ß inactivation synergistically protect against obesity, insulin resistance and liver steatosis in mice.
Am J Transplant
; 21(8): 2688-2697, 2021 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-33370494
2.
A novel model of reno-cardiac syndrome in the C57BL/ 6 mouse strain.
BMC Nephrol
; 19(1): 346, 2018 12 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-30509210
3.
Inactivation of class II PI3K-C2α induces leptin resistance, age-dependent insulin resistance and obesity in male mice.
Diabetologia
; 59(7): 1503-1512, 2016 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-27138914
4.
Local synthesis of the phosphatidylinositol-3,4-bisphosphate lipid drives focal adhesion turnover.
Dev Cell
; 57(14): 1694-1711.e7, 2022 07 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-35809565
5.
Constitutively active Akt1 expression in mouse pancreas requires S6 kinase 1 for insulinoma formation.
J Clin Invest
; 118(11): 3629-38, 2008 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-18846252
6.
Immunohistochemistry of Kidney a-SMA, Collagen 1, and Collagen 3, in A Novel Mouse Model of Reno-cardiac Syndrome.
Bio Protoc
; 10(18): e3751, 2020 Sep 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-33659410
7.
Vps34 PI 3-kinase inactivation enhances insulin sensitivity through reprogramming of mitochondrial metabolism.
Nat Commun
; 8(1): 1804, 2017 11 27.
Artigo
em Inglês
| MEDLINE | ID: mdl-29180704
8.
Roles of the lactogens and somatogens in perinatal and postnatal metabolism and growth: studies of a novel mouse model combining lactogen resistance and growth hormone deficiency.
Endocrinology
; 146(1): 103-12, 2005 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-15388648
9.
Inactivation of the Class II PI3K-C2ß Potentiates Insulin Signaling and Sensitivity.
Cell Rep
; 13(9): 1881-94, 2015 Dec 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26655903
10.
The combined deletion of S6K1 and Akt2 deteriorates glycemic control in a high-fat diet.
Mol Cell Biol
; 32(19): 4001-11, 2012 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-22851690
11.
S6 kinase deletion suppresses muscle growth adaptations to nutrient availability by activating AMP kinase.
Cell Metab
; 5(6): 476-87, 2007 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-17550782