Detalhe da pesquisa
1.
ABCG2-Expressing Clonal Repopulating Endothelial Cells Serve to Form and Maintain Blood Vessels.
Circulation
; 2024 Apr 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-38682338
2.
Impaired hemodynamic renal reserve response following recovery from established acute kidney injury and improvement by hydrodynamic isotonic fluid delivery.
Am J Physiol Renal Physiol
; 326(1): F86-F94, 2024 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37881874
3.
Macrophage dynamics in kidney repair: elucidation of a COX-2-dependent MafB pathway to affect macrophage differentiation.
Kidney Int
; 101(1): 15-18, 2022 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-34991803
4.
Serum IL-17 levels are higher in critically ill patients with AKI and associated with worse outcomes.
Crit Care
; 26(1): 107, 2022 04 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-35422004
5.
Contribution of Th17 cells to tissue injury in hypertension.
Curr Opin Nephrol Hypertens
; 30(2): 151-158, 2021 03 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33394732
6.
Mutation of RORγT reveals a role for Th17 cells in both injury and recovery from renal ischemia-reperfusion injury.
Am J Physiol Renal Physiol
; 319(5): F796-F808, 2020 11 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-32924545
7.
Beast of (renal) burden? Bst1-expressing neutrophils in kidney injury.
Am J Physiol Renal Physiol
; 326(2): F165-F166, 2024 02 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-38095024
8.
Exogenous Gene Transmission of Isocitrate Dehydrogenase 2 Mimics Ischemic Preconditioning Protection.
J Am Soc Nephrol
; 29(4): 1154-1164, 2018 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-29371417
9.
Th17 cells contribute to pulmonary fibrosis and inflammation during chronic kidney disease progression after acute ischemia.
Am J Physiol Regul Integr Comp Physiol
; 314(2): R265-R273, 2018 02 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-29118018
10.
Crystals or His(stones): Rethinking AKI in Tumor Lysis Syndrome.
J Am Soc Nephrol
; 33(6): 1055-1057, 2022 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-35641305
11.
Hydrodynamic Isotonic Fluid Delivery Ameliorates Moderate-to-Severe Ischemia-Reperfusion Injury in Rat Kidneys.
J Am Soc Nephrol
; 28(7): 2081-2092, 2017 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-28122967
12.
Human adipose stromal cell therapy improves survival and reduces renal inflammation and capillary rarefaction in acute kidney injury.
J Cell Mol Med
; 21(7): 1420-1430, 2017 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-28455887
13.
Endothelial colony-forming cells ameliorate endothelial dysfunction via secreted factors following ischemia-reperfusion injury.
Am J Physiol Renal Physiol
; 312(5): F897-F907, 2017 05 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-28228404
14.
IL-17 mediates neutrophil infiltration and renal fibrosis following recovery from ischemia reperfusion: compensatory role of natural killer cells in athymic rats.
Am J Physiol Renal Physiol
; 312(3): F385-F397, 2017 03 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-27852609
15.
Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.
J Am Soc Nephrol
; 27(3): 687-97, 2016 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-26519085
16.
Th-17 cell activation in response to high salt following acute kidney injury is associated with progressive fibrosis and attenuated by AT-1R antagonism.
Kidney Int
; 88(4): 776-84, 2015 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-26200947
17.
Effect of renal shock wave lithotripsy on the development of metabolic syndrome in a juvenile swine model: a pilot study.
J Urol
; 193(4): 1409-16, 2015 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-25245490
18.
Circulating and tissue resident endothelial progenitor cells.
J Cell Physiol
; 229(1): 10-6, 2014 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-23794280
19.
The case for capillary rarefaction in the AKI to CKD progression: insights from multiple injury models.
Am J Physiol Renal Physiol
; 317(5): F1253-F1254, 2019 11 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-31630545
20.
Unique Gene Expression in Developing Ascending Vasa Recta: A Tale of Tie.
J Am Soc Nephrol
; 29(4): 1073-1074, 2018 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-29531096