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J Med Chem ; 51(9): 2734-43, 2008 May 08.
Artigo em Inglês | MEDLINE | ID: mdl-18393399

RESUMO

Type 1 interferons (IFN) bind specifically to the corresponding receptor, IFNAR. Agonists and antagonists for IFNAR have potential therapeutic value in the treatment of viral infections and systemic lupus erythematosus, respectively. Specific sequences on the surface of IFN, IFN receptor recognition peptides (IRRPs) mediate the binding and signal transduction when IFN interacts with IFNAR. Structural features of two such IRRPs, IRRP-1 and IRRP-3, were used as templates to design small molecule mimetics. In silico screening was used to identify the molecular structural features mimicking their surface characteristics. A set of 26 compounds were synthesized and their ability to interfere with IFN-IFNAR interactions was investigated. Two compounds exhibited antagonist activity, specifically, blocking IFN-inducible Stat phosphorylation Stat complex-DNA binding. Design principles revealed here pave the way toward a novel series of small molecules as antagonists for IFN-IFNAR interactions.


Assuntos
Interferon-alfa/química , Modelos Moleculares , Peptídeos/química , Receptor de Interferon alfa e beta/antagonistas & inibidores , Receptor de Interferon alfa e beta/metabolismo , Linhagem Celular Tumoral , DNA/metabolismo , Desenho de Fármacos , Furanos/síntese química , Furanos/química , Furanos/farmacologia , Guanidinas/síntese química , Guanidinas/química , Guanidinas/farmacologia , Humanos , Interferon-alfa/metabolismo , Mimetismo Molecular , Fosforilação , Pirimidinas/síntese química , Pirimidinas/química , Pirimidinas/farmacologia , Fator de Transcrição STAT1/metabolismo , Relação Estrutura-Atividade , Tiofenos/síntese química , Tiofenos/química , Tiofenos/farmacologia
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