Detalhe da pesquisa
1.
Connexin-43 hemichannels orchestrate NOD-like receptor protein-3 (NLRP3) inflammasome activation and sterile inflammation in tubular injury.
Cell Commun Signal
; 21(1): 263, 2023 09 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-37770948
2.
The Angiogenesis Inhibitor Isthmin-1 (ISM1) Is Overexpressed in Experimental Models of Glomerulopathy and Impairs the Viability of Podocytes.
Int J Mol Sci
; 24(3)2023 Feb 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-36769045
3.
Genetic inactivation of Semaphorin 3C protects mice from acute kidney injury.
Kidney Int
; 101(4): 720-732, 2022 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-35090878
4.
The Role of Connexin 43 in Renal Disease: Insights from In Vivo Models of Experimental Nephropathy.
Int J Mol Sci
; 23(21)2022 Oct 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-36361888
5.
Galectin-3 in Kidney Diseases: From an Old Protein to a New Therapeutic Target.
Int J Mol Sci
; 23(6)2022 Mar 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-35328545
6.
Deletion of Notch3 Impairs Contractility of Renal Resistance Vessels Due to Deficient Ca2+ Entry.
Int J Mol Sci
; 23(24)2022 Dec 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-36555708
7.
Connexin 43: A Target for the Treatment of Inflammation in Secondary Complications of the Kidney and Eye in Diabetes.
Int J Mol Sci
; 23(2)2022 Jan 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-35054783
8.
Activation of Notch3 in Renal Tubular Cells Leads to Progressive Cystic Kidney Disease.
Int J Mol Sci
; 23(2)2022 Jan 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-35055068
9.
Super-resolved local recruitment of CLDN5 to filtration slits implicates a direct relationship with podocyte foot process effacement.
J Cell Mol Med
; 25(16): 7631-7641, 2021 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-34156149
10.
Periostin Promotes Cell Proliferation and Macrophage Polarization to Drive Repair after AKI.
J Am Soc Nephrol
; 31(1): 85-100, 2020 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-31690575
11.
Blocking Connexin-43 mediated hemichannel activity protects against early tubular injury in experimental chronic kidney disease.
Cell Commun Signal
; 18(1): 79, 2020 05 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-32450899
12.
The Role of Palladin in Podocytes.
J Am Soc Nephrol
; 29(6): 1662-1678, 2018 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-29720549
13.
Notch3 orchestrates epithelial and inflammatory responses to promote acute kidney injury.
Kidney Int
; 94(1): 126-138, 2018 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-29751972
14.
Connexin 43: a New Therapeutic Target Against Chronic Kidney Disease.
Cell Physiol Biochem
; 49(3): 985, 2018.
Artigo
em Inglês
| MEDLINE | ID: mdl-30196298
15.
NFκB-Induced Periostin Activates Integrin-ß3 Signaling to Promote Renal Injury in GN.
J Am Soc Nephrol
; 28(5): 1475-1490, 2017 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-27920156
16.
Decreased Expression of Connexin 43 Blunts the Progression of Experimental GN.
J Am Soc Nephrol
; 28(10): 2915-2930, 2017 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-28667079
17.
Functional roles of connexins and pannexins in the kidney.
Cell Mol Life Sci
; 72(15): 2869-77, 2015 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-26082183
18.
Discoidin domain receptor-1 and periostin: new players in chronic kidney disease.
Nephrol Dial Transplant
; 30(12): 1965-71, 2015 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-25829327
19.
MiR-21 is up-regulated in urinary exosomes of chronic kidney disease patients and after glomerular injury.
J Cell Mol Med
; 23(7): 4839-4843, 2019 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-31066165
20.
Targeting connexin 43 protects against the progression of experimental chronic kidney disease in mice.
Kidney Int
; 86(4): 768-79, 2014 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-24850151