Detalhe da pesquisa
1.
The role of HLA-DQ8 beta57 polymorphism in the anti-gluten T-cell response in coeliac disease.
Nature
; 456(7221): 534-8, 2008 Nov 27.
Artigo
em Inglês
| MEDLINE | ID: mdl-19037317
2.
Janus kinase-2 inhibition induces durable tolerance to alloantigen by human dendritic cell-stimulated T cells yet preserves immunity to recall antigen.
Blood
; 118(19): 5330-9, 2011 Nov 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-21917753
3.
Mechanisms imposing the Vbeta bias of Valpha14 natural killer T cells and consequences for microbial glycolipid recognition.
J Exp Med
; 203(5): 1197-207, 2006 May 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-16651387
4.
Reprogramming of CTLs into natural killer-like cells in celiac disease.
J Exp Med
; 203(5): 1343-55, 2006 May 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-16682498
5.
Primary T Cells from Cutaneous T-cell Lymphoma Skin Explants Display an Exhausted Immune Checkpoint Profile.
Cancer Immunol Res
; 6(8): 900-909, 2018 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-29895574
6.
Human Dendritic Cells Mitigate NK-Cell Dysfunction Mediated by Nonselective JAK1/2 Blockade.
Cancer Immunol Res
; 5(1): 52-60, 2017 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-27923824
7.
T-cell Exhaustion in Multiple Myeloma Relapse after Autotransplant: Optimal Timing of Immunotherapy.
Cancer Immunol Res
; 4(1): 61-71, 2016 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-26464015
8.
Phenotypic and functional activation of hyporesponsive KIRnegNKG2Aneg human NK-cell precursors requires IL12p70 provided by Poly(I:C)-matured monocyte-derived dendritic cells.
Cancer Immunol Res
; 2(10): 1000-10, 2014 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-25023628
9.
Interleukin 15 primes natural killer cells to kill via NKG2D and cPLA2 and this pathway is active in psoriatic arthritis.
PLoS One
; 8(9): e76292, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-24086722
10.
Nucleotide sequencing of psoriatic arthritis tissue before and during methotrexate administration reveals a complex inflammatory T cell infiltrate with very few clones exhibiting features that suggest they drive the inflammatory process by recognizing autoantigens.
J Immunol
; 172(3): 1935-44, 2004 Feb 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-14734779