Detalhe da pesquisa
1.
CCR5 knockout prevents neuronal injury and behavioral impairment induced in a transgenic mouse model by a CXCR4-using HIV-1 glycoprotein 120.
J Immunol
; 193(4): 1895-910, 2014 Aug 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-25031461
2.
Activation of p38 MAPK is required in monocytic and neuronal cells for HIV glycoprotein 120-induced neurotoxicity.
J Immunol
; 185(8): 4883-95, 2010 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-20855878
3.
An Alzheimer's disease-relevant presenilin-1 mutation augments amyloid-beta-induced oligodendrocyte dysfunction.
Glia
; 59(4): 627-40, 2011 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-21294162
4.
Early oligodendrocyte/myelin pathology in Alzheimer's disease mice constitutes a novel therapeutic target.
Am J Pathol
; 177(3): 1422-35, 2010 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-20696774
5.
Triple-transgenic Alzheimer's disease mice exhibit region-specific abnormalities in brain myelination patterns prior to appearance of amyloid and tau pathology.
Glia
; 57(1): 54-65, 2009 Jan 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-18661556
6.
Genetic knockouts suggest a critical role for HIV co-receptors in models of HIV gp120-induced brain injury.
J Neuroimmune Pharmacol
; 7(2): 306-18, 2012 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-22124968