Detalhe da pesquisa
1.
Insights into the etiology and physiopathology of MODY5/HNF1B pancreatic phenotype with a mouse model of the human disease.
J Pathol
; 254(1): 31-45, 2021 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-33527355
2.
Cadherin-1 and cadherin-3 cooperation determines the aggressiveness of pancreatic ductal adenocarcinoma.
Br J Cancer
; 118(4): 546-557, 2018 02 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-29161242
3.
Missense mutations in SLC26A8, encoding a sperm-specific activator of CFTR, are associated with human asthenozoospermia.
Am J Hum Genet
; 92(5): 760-6, 2013 May 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-23582645
4.
The testis anion transporter TAT1 (SLC26A8) physically and functionally interacts with the cystic fibrosis transmembrane conductance regulator channel: a potential role during sperm capacitation.
Hum Mol Genet
; 21(6): 1287-98, 2012 Mar 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-22121115
5.
Structural Basis of PML-RARA Oncoprotein Targeting by Arsenic Unravels a Cysteine Rheostat Controlling PML Body Assembly and Function.
Cancer Discov
; 13(12): 2548-2565, 2023 12 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-37655965
6.
Pancreatic Ductal Deletion of Hnf1b Disrupts Exocrine Homeostasis, Leads to Pancreatitis, and Facilitates Tumorigenesis.
Cell Mol Gastroenterol Hepatol
; 8(3): 487-511, 2019.
Artigo
em Inglês
| MEDLINE | ID: mdl-31229598
7.
Assessment of the frequency of sperm annulus defects in a large cohort of patients presenting asthenozoospermia.
Basic Clin Androl
; 25: 10, 2015.
Artigo
em Inglês
| MEDLINE | ID: mdl-26576287