Detalhe da pesquisa
1.
Neuronal expression of GalNAc transferase is sufficient to prevent the age-related neurodegenerative phenotype of complex ganglioside-deficient mice.
J Neurosci
; 34(3): 880-91, 2014 Jan 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-24431446
2.
The pre-synaptic motor nerve terminal as a site for antibody-mediated neurotoxicity in autoimmune neuropathies and synaptopathies.
J Anat
; 224(1): 36-44, 2014 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-23937354
3.
A mammalian fatty acid hydroxylase responsible for the formation of alpha-hydroxylated galactosylceramide in myelin.
Biochem J
; 388(Pt 1): 245-54, 2005 May 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-15658937
4.
C1q-targeted inhibition of the classical complement pathway prevents injury in a novel mouse model of acute motor axonal neuropathy.
Acta Neuropathol Commun
; 4: 23, 2016 Mar 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-26936605
5.
Anti-ganglioside antibody internalization attenuates motor nerve terminal injury in a mouse model of acute motor axonal neuropathy.
J Clin Invest
; 122(3): 1037-51, 2012 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-22307327
6.
Reversal of non-hydroxy:alpha-hydroxy galactosylceramide ratio and unstable myelin in transgenic mice overexpressing UDP-galactose:ceramide galactosyltransferase.
J Neurochem
; 94(2): 469-81, 2005 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-15998297
7.
N-glycosylation is required for full enzymic activity of the murine galactosylceramide sulphotransferase.
Biochem J
; 368(Pt 1): 317-24, 2002 Nov 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-12175333