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Pharmacogenomics J ; 18(2): 270-274, 2018 04.
Artigo em Inglês | MEDLINE | ID: mdl-28534526

RESUMO

Neutropenia and infection are major dose-limiting side effects of chemotherapy. The risk of initial infection and subsequent complications are directly related to the depth and duration of neutropenia. Recent genome-wide association studies identified variants in DARC and CXCL2 genes, and in ORMDL3-GSDMA-CSF3 locus on chromosome 17q21 that influence white blood cell and neutrophil counts in healthy individuals. To investigate whether polymorphisms in these loci in conjunction with chemotherapy may modulate risk of treatment complications, we analyzed 21 SNPs across these genes for an association with chemotherapy-related neutropenia and infection in 286 Caucasian children with acute lymphoblastic leukemia. After correction for multiple testing, DARC polymorphism rs3027012 in 5'-UTR was associated with higher risk of low absolute phagocyte count (APC<500 and <1000 cells per microliter, P=0.001 and P<0.0005, respectively) and hospitalization due to febrile neutropenia (P=0.002). Protective effect was instead seen for DARC rs12075 A to G substitution (P=0.004). The SNP rs3859192 in the GSDMA were associated with hospitalization due to infection (P=0.004); infection was also modulated in the additive manner by the CXCL2 rs16850408 (P=0.002). This study shows for the first time that the variations in DARC, GSDMA and CXCL2 genes may play a role in the onset of chemotherapy complications.


Assuntos
Antineoplásicos/efeitos adversos , Neutropenia/genética , Polimorfismo de Nucleotídeo Único/genética , Leucemia-Linfoma Linfoblástico de Células Precursoras/genética , Antineoplásicos/sangue , Quimiocina CXCL2/genética , Criança , Sistema do Grupo Sanguíneo Duffy/genética , Humanos , Contagem de Leucócitos/tendências , Proteínas de Neoplasias/genética , Neutropenia/sangue , Neutropenia/induzido quimicamente , Neutrófilos/efeitos dos fármacos , Neutrófilos/fisiologia , Variantes Farmacogenômicos/efeitos dos fármacos , Variantes Farmacogenômicos/genética , Leucemia-Linfoma Linfoblástico de Células Precursoras/sangue , Leucemia-Linfoma Linfoblástico de Células Precursoras/tratamento farmacológico , Receptores de Superfície Celular/genética
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