Detalhe da pesquisa
1.
Connexin 43 modulates reverse electron transfer in cardiac mitochondria from inducible knock-out Cx43Cre-ER(T)/fl mice by altering the coenzyme Q pool.
Basic Res Cardiol
; 2024 May 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-38724619
2.
Long-Term Protective Effects of Succinate Dehydrogenase Inhibition during Reperfusion with Malonate on Post-Infarction Left Ventricular Scar and Remodeling in Mice.
Int J Mol Sci
; 25(8)2024 Apr 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-38673951
3.
TRPV4 Channels Promote Pathological, but Not Physiological, Cardiac Remodeling through the Activation of Calcineurin/NFAT and TRPC6.
Int J Mol Sci
; 25(3)2024 Jan 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-38338818
4.
Calpains as Potential Therapeutic Targets for Myocardial Hypertrophy.
Int J Mol Sci
; 23(8)2022 Apr 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-35456920
5.
Effect of COMBinAtion therapy with remote ischemic conditioning and exenatide on the Myocardial Infarct size: a two-by-two factorial randomized trial (COMBAT-MI).
Basic Res Cardiol
; 116(1): 4, 2021 01 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-33495853
6.
Citric Acid Cycle Metabolites Predict Infarct Size in Pigs Submitted to Transient Coronary Artery Occlusion and Treated with Succinate Dehydrogenase Inhibitors or Remote Ischemic Perconditioning.
Int J Mol Sci
; 22(8)2021 Apr 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-33923786
7.
Aging Impairs Reverse Remodeling and Recovery of Ventricular Function after Isoproterenol-Induced Cardiomyopathy.
Int J Mol Sci
; 23(1)2021 Dec 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-35008601
8.
High-fat diet improves tolerance to myocardial ischemia by delaying normalization of intracellular PH at reperfusion.
J Mol Cell Cardiol
; 133: 164-173, 2019 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-31194987
9.
Calpains mediate isoproterenol-induced hypertrophy through modulation of GRK2.
Basic Res Cardiol
; 114(3): 21, 2019 03 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-30915659
10.
Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling.
Basic Res Cardiol
; 111(2): 18, 2016 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-26924441
11.
Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discovery.
Basic Res Cardiol
; 111(6): 70, 2016 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-27766474
12.
Obesity induced by high fat diet attenuates postinfarct myocardial remodeling and dysfunction in adult B6D2F1 mice.
J Mol Cell Cardiol
; 84: 154-61, 2015 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-25953257
13.
Microtubule stabilization with paclitaxel does not protect against infarction in isolated rat hearts.
Exp Physiol
; 100(1): 23-34, 2015 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-25557728
14.
Ischemic preconditioning protects cardiomyocyte mitochondria through mechanisms independent of cytosol.
J Mol Cell Cardiol
; 68: 79-88, 2014 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-24434643
15.
Role of the parasympathetic nervous system in cardioprotection by remote hindlimb ischaemic preconditioning.
Exp Physiol
; 98(2): 425-34, 2013 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-22872660
16.
Spontaneous reperfusion enhances succinate concentration in peripheral blood from stemi patients but its levels does not correlate with myocardial infarct size or area at risk.
Sci Rep
; 13(1): 6907, 2023 04 27.
Artigo
em Inglês
| MEDLINE | ID: mdl-37106099
17.
Myocardial edema: a translational view.
J Mol Cell Cardiol
; 52(5): 931-9, 2012 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-22285721
18.
Cardiac fibroblasts display endurance to ischemia, high ROS control and elevated respiration regulated by the JAK2/STAT pathway.
FEBS J
; 289(9): 2540-2561, 2022 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-34796659
19.
Defective dimerization of FoF1-ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging.
Aging Cell
; 21(3): e13564, 2022 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-35233924
20.
cGMP/PKG pathway mediates myocardial postconditioning protection in rat hearts by delaying normalization of intracellular acidosis during reperfusion.
J Mol Cell Cardiol
; 50(5): 903-9, 2011 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-21362429