Evidence for a Northern Hemispheric trigger of the 100,000-y glacial cyclicity.

The causes of the Mid-Pleistocene Transition, the shift from ∼41-ky to 100-ky interglacial-glacial cycles and more intense ice ages, remain intensely debated, as this fundamental change occurred between ∼1,250 and 650 ka without substantial changes in astronomical climate forcings. Recent studies disagree about the relative importance of events and processes in the Northern and Southern Hemispheres, as well as whether the shift occurred gradually over several interglacial-glacial cycles or abruptly at ∼900 ka. We address these issues using a north-to-south reconstruction of the Atlantic arm of the global meridional overturning ocean circulation, a primary means for distributing heat around the globe, using neodymium (Nd) isotopes. Results reveal a period of intense erosion affecting the cratonic shields surrounding the North Atlantic between Marine Isotope Stages (MIS) 27 and 25 (∼980 and 950 ka), reflected by unusually low Nd isotope ratios in deep North Atlantic seawater. This episode preceded a major ocean circulation weakening between MIS 25 and 21 (950 and 860 ka) that coincided with the first ∼100-ky-long interglacial-glacial onset of Northern Hemisphere glaciation at around 2.4 to 2.8 Ma. The data point to a Northern Hemisphere-sourced initiation for the transition, possibly induced through regolith loss and increased exposure of the crystalline bedrock, which would lead to increased friction, enabling larger ice sheets that are characteristic of the 100-ky interglacial-glacial cycles.

Causes and timing of recurring subarctic Pacific hypoxia.

Several North Pacific studies of the last deglaciation show hypoxia throughout the ocean margins and attribute this phenomenon to the effects of abrupt warming and meltwater inputs. Yet, because of the lack of long records spanning multiple glacial cycles and deglaciation events, it is unclear whether deoxygenation was a regular occurrence of warming events and whether deglaciation and/or other conditions promoted hypoxia throughout time. Here, subarctic Pacific laminated sediments from the past 1.2 million years demonstrate that hypoxic events recurred throughout the Pleistocene as episodes of highly productive phytoplankton growth and were generally associated with interglacial climates, high sea levels, and enhanced nitrate utilization-but not with deglaciations. We suggest that hypoxia was typically stimulated by high productivity from iron fertilization facilitated by redox-remobilized iron from flooded continental shelves.