Detalhe da pesquisa
1.
H+-slip correlated to rotor free-wheeling as cause of F1FO-ATPase dysfunction in primary mitochondrial disorders.
Med Res Rev
; 44(3): 1183-1188, 2024 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-38167815
2.
Emerging Roles for the Mitochondrial ATP Synthase Supercomplexes.
Trends Biochem Sci
; 44(10): 821-823, 2019 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-31402189
3.
Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions.
J Transl Med
; 21(1): 757, 2023 10 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-37884933
4.
1,5-disubstituted-1,2,3-triazoles counteract mitochondrial dysfunction acting on F1FO-ATPase in models of cardiovascular diseases.
Pharmacol Res
; 187: 106561, 2023 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-36410676
5.
Effects of cryopreservation on the mitochondrial bioenergetics of bovine sperm.
Reprod Domest Anim
; 58(1): 184-188, 2023 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-36107136
6.
Inflammation, Mitochondria and Natural Compounds Together in the Circle of Trust.
Int J Mol Sci
; 24(7)2023 Mar 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-37047080
7.
Cell Metabolism Therapy by Small Natural Compounds.
Int J Mol Sci
; 24(18)2023 Sep 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-37762081
8.
A Lethal Channel between the ATP Synthase Monomers.
Trends Biochem Sci
; 43(5): 311-313, 2018 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-29555114
9.
Mitochondrial F-type ATP synthase: multiple enzyme functions revealed by the membrane-embedded FO structure.
Crit Rev Biochem Mol Biol
; 55(4): 309-321, 2020 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-32580582
10.
Protein folding and unfolding: proline cis-trans isomerization at the c subunits of F1 FO -ATPase might open a high conductance ion channel.
Proteins
; 90(11): 2001-2005, 2022 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-35532281
11.
Cellular Metabolism and Bioenergetic Function in Human Fibroblasts and Preadipocytes of Type 2 Familial Partial Lipodystrophy.
Int J Mol Sci
; 23(15)2022 Aug 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-35955791
12.
The Impairment of Cell Metabolism by Cardiovascular Toxicity of Doxorubicin Is Reversed by Bergamot Polyphenolic Fraction Treatment in Endothelial Cells.
Int J Mol Sci
; 23(16)2022 Aug 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-36012238
13.
Ca2+ as cofactor of the mitochondrial H+ -translocating F1 FO -ATP(hydrol)ase.
Proteins
; 89(5): 477-482, 2021 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-33378096
14.
Relationship between serum concentration, functional parameters and cell bioenergetics in IPEC-J2 cell line.
Histochem Cell Biol
; 156(1): 59-67, 2021 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-33725198
15.
The mitochondrial F1FO-ATPase exploits the dithiol redox state to modulate the permeability transition pore.
Arch Biochem Biophys
; 712: 109027, 2021 11 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-34520732
16.
Sulfide affects the mitochondrial respiration, the Ca2+-activated F1FO-ATPase activity and the permeability transition pore but does not change the Mg2+-activated F1FO-ATPase activity in swine heart mitochondria.
Pharmacol Res
; 166: 105495, 2021 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-33600941
17.
The mitochondrial permeability transition pore in cell death: A promising drug binding bioarchitecture.
Med Res Rev
; 40(2): 811-817, 2020 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-31617227
18.
Phenylglyoxal inhibition of the mitochondrial F1FO-ATPase activated by Mg2+ or by Ca2+ provides clues on the mitochondrial permeability transition pore.
Arch Biochem Biophys
; 681: 108258, 2020 03 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-31917961
19.
Effects of Hydrogen Sulfide Donor NaHS on Porcine Vascular Wall-Mesenchymal Stem Cells.
Int J Mol Sci
; 21(15)2020 Jul 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-32722269
20.
Characterization of metabolic profiles and lipopolysaccharide effects on porcine vascular wall mesenchymal stem cells.
J Cell Physiol
; 234(10): 16685-16691, 2019 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-30825197