Detalhe da pesquisa
1.
Hypoxia Inducible Factor-1α binds and activates γ-secretase for Aß production under hypoxia and cerebral hypoperfusion.
Mol Psychiatry
; 27(10): 4264-4273, 2022 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-35764706
2.
tPA Deficiency Underlies Neurovascular Coupling Dysfunction by Amyloid-ß.
J Neurosci
; 40(42): 8160-8173, 2020 10 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-32928888
3.
Brain Perivascular Macrophages Initiate the Neurovascular Dysfunction of Alzheimer Aß Peptides.
Circ Res
; 121(3): 258-269, 2017 Jul 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-28515043
4.
Endothelial Dysfunction and Amyloid-ß-Induced Neurovascular Alterations.
Cell Mol Neurobiol
; 36(2): 155-65, 2016 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-26328781
5.
Innate immunity receptor CD36 promotes cerebral amyloid angiopathy.
Proc Natl Acad Sci U S A
; 110(8): 3089-94, 2013 Feb 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-23382216
6.
Age-dependent neurovascular dysfunction and damage in a mouse model of cerebral amyloid angiopathy.
Stroke
; 45(6): 1815-21, 2014 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-24781082
7.
Prohibitin viral gene transfer protects hippocampal CA1 neurons from ischemia and ameliorates postischemic hippocampal dysfunction.
Stroke
; 45(4): 1131-8, 2014 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-24619393
8.
Scavenger receptor CD36 is essential for the cerebrovascular oxidative stress and neurovascular dysfunction induced by amyloid-beta.
Proc Natl Acad Sci U S A
; 108(12): 5063-8, 2011 Mar 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-21383152
9.
Meningeal interleukin-17-producing T cells mediate cognitive impairment in a mouse model of salt-sensitive hypertension.
Nat Neurosci
; 27(1): 63-77, 2024 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-38049579
10.
Brain and circulating levels of Aß1-40 differentially contribute to vasomotor dysfunction in the mouse brain.
Stroke
; 44(1): 198-204, 2013 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-23204056
11.
Prostaglandin E2 EP1 receptors: downstream effectors of COX-2 neurotoxicity.
Nat Med
; 12(2): 225-9, 2006 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-16432513
12.
Border-associated macrophages promote cerebral amyloid angiopathy and cognitive impairment through vascular oxidative stress.
Res Sq
; 2023 Apr 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-37162996
13.
Hippocampal glial inflammatory markers are differentially altered in a novel mouse model of perimenopausal cerebral amyloid angiopathy.
Front Aging Neurosci
; 15: 1280218, 2023.
Artigo
em Inglês
| MEDLINE | ID: mdl-38035277
14.
Cell autonomous role of border associated macrophages in ApoE4 neurovascular dysfunction and susceptibility to white matter injury.
Res Sq
; 2023 Aug 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-37577565
15.
Border-associated macrophages promote cerebral amyloid angiopathy and cognitive impairment through vascular oxidative stress.
Mol Neurodegener
; 18(1): 73, 2023 10 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-37789345
16.
Vascular oxidative stress causes neutrophil arrest in brain capillaries, leading to decreased cerebral blood flow and contributing to memory impairment in a mouse model of Alzheimerâ™s disease.
bioRxiv
; 2023 Feb 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-36824768
17.
Angiotensin II type 2 receptor-coupled nitric oxide production modulates free radical availability and voltage-gated Ca2+ currents in NTS neurons.
Am J Physiol Regul Integr Comp Physiol
; 302(9): R1076-83, 2012 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-22378773
18.
Chronic intermittent hypoxia induces NMDA receptor-dependent plasticity and suppresses nitric oxide signaling in the mouse hypothalamic paraventricular nucleus.
J Neurosci
; 30(36): 12103-12, 2010 Sep 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-20826673
19.
The cerebrovascular dysfunction induced by slow pressor doses of angiotensin II precedes the development of hypertension.
Am J Physiol Heart Circ Physiol
; 300(1): H397-407, 2011 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-20971763
20.
Key role of tissue plasminogen activator in neurovascular coupling.
Proc Natl Acad Sci U S A
; 105(3): 1073-8, 2008 Jan 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-18195371