Detalhe da pesquisa
1.
Virus-induced senescence is a driver and therapeutic target in COVID-19.
Nature
; 599(7884): 283-289, 2021 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-34517409
2.
Senescence-associated reprogramming promotes cancer stemness.
Nature
; 553(7686): 96-100, 2018 01 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-29258294
3.
Adaptive T-cell immunity controls senescence-prone MyD88- or CARD11-mutant B-cell lymphomas.
Blood
; 137(20): 2785-2799, 2021 05 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-33232972
4.
Pharmacological restoration and therapeutic targeting of the B-cell phenotype in classical Hodgkin lymphoma.
Blood
; 129(1): 71-81, 2017 01 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-27733358
5.
Synthetic lethal metabolic targeting of cellular senescence in cancer therapy.
Nature
; 501(7467): 421-5, 2013 Sep 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-23945590
6.
Opposing roles of NF-κB in anti-cancer treatment outcome unveiled by cross-species investigations.
Genes Dev
; 25(20): 2137-46, 2011 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-21979374
7.
B-cell-specific conditional expression of Myd88p.L252P leads to the development of diffuse large B-cell lymphoma in mice.
Blood
; 127(22): 2732-41, 2016 06 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-27048211
8.
Combined inhibition of PI3K-related DNA damage response kinases and mTORC1 induces apoptosis in MYC-driven B-cell lymphomas.
Blood
; 121(15): 2964-74, 2013 Apr 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-23403624
9.
Cellular senescence: Neither irreversible nor reversible.
J Exp Med
; 221(4)2024 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-38385946
10.
The Myc/macrophage tango: oncogene-induced senescence, Myc style.
Semin Cancer Biol
; 21(6): 377-84, 2011 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-22019769
11.
An Autochthonous Mouse Model of Myd88- and BCL2-Driven Diffuse Large B-cell Lymphoma Reveals Actionable Molecular Vulnerabilities.
Blood Cancer Discov
; 2(1): 70-91, 2021 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33447829
12.
H3K9me3-mediated epigenetic regulation of senescence in mice predicts outcome of lymphoma patients.
Nat Commun
; 11(1): 3651, 2020 07 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-32686676
13.
Targeting the Senescence-Overriding Cooperative Activity of Structurally Unrelated H3K9 Demethylases in Melanoma.
Cancer Cell
; 33(2): 322-336.e8, 2018 02 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-29438700
14.
Targeting the Senescence-Overriding Cooperative Activity of Structurally Unrelated H3K9 Demethylases in Melanoma.
Cancer Cell
; 33(4): 785, 2018 04 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-29634951
15.
Non-cell-autonomous tumor suppression: oncogene-provoked apoptosis promotes tumor cell senescence via stromal crosstalk.
J Mol Med (Berl)
; 89(9): 869-75, 2011 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-21594578
16.
Tumor stroma-derived TGF-beta limits myc-driven lymphomagenesis via Suv39h1-dependent senescence.
Cancer Cell
; 17(3): 262-72, 2010 Mar 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-20227040
17.
The Myc-evoked DNA damage response accounts for treatment resistance in primary lymphomas in vivo.
Blood
; 110(8): 2996-3004, 2007 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-17562874
18.
Analysis of p53 "latency" and "activation" by fluorescence correlation spectroscopy. Evidence for different modes of high affinity DNA binding.
J Biol Chem
; 278(35): 32587-95, 2003 Aug 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-12813031
19.
Specific interaction of p53 with target binding sites is determined by DNA conformation and is regulated by the C-terminal domain.
J Biol Chem
; 277(43): 41192-203, 2002 Oct 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-12171916