RESUMO
The T immunity system in patients with isolated and combined craniocerebral trauma is characterized during the course of the posttraumatic period. The revealed T lymphocytopenia and decrease in the fraction of the immunoregulating cells are, on the one hand, the cause of the frequent inflammatory complications and, on the other, conducive to the development of autosensitization of the organism to the brain antigens, which was confirmed in study of the specific features of delayed hypersensitivity.
Assuntos
Concussão Encefálica/imunologia , Encéfalo/imunologia , Linfócitos T/imunologia , Adolescente , Adulto , Lesões Encefálicas/complicações , Inibição de Migração Celular , Humanos , Hipersensibilidade Tardia/etiologia , Contagem de Leucócitos , Pessoa de Meia-Idade , Formação de Roseta , Linfócitos T Auxiliares-Indutores , Linfócitos T ReguladoresRESUMO
Bitemporal UHF electric field is shown to enhance glucocorticoid adrenal function unlike inhibition of the thyroid function suppressing a primary immune response (PIR) in the productive phase. The combined exposure to bitemporal UHF electric field and decimeter waves of the adrenals doubles glucocorticoid synthesis abolishing the inhibitory action of the UHF therapy on thyroid function resultant in much more suppressed PIR. Both modalities inhibit thymic production. Decimeter waves alone are less effective. The exposure of the thyroid to decimeter waves initiated PIR by 2.5-fold activation of medullar lymphocytes and by a 80% increase in the thymic function. No response was achieved in combined action on the thyroid of the electric field and decimeter waves.