1.
Genetics
; 199(1): 151-6, 2015 Jan.
Artigo
em Inglês
| MEDLINE
| ID: mdl-25339611
RESUMO
We show that loss-of-function mutations in kinases of the MLK-1 pathway (mlk-1, mek-1, and kgb-1/jnk) function cell-autonomously in neurons to suppress defects in synapse formation and axon termination caused by rpm-1 loss of function. Our genetic analysis also suggests that the phosphatase PPM-1, like RPM-1, is a potential inhibitor of kinases in the MLK-1 pathway.