Detalhe da pesquisa
1.
Suppression of PKR promotes network excitability and enhanced cognition by interferon-γ-mediated disinhibition.
Cell
; 147(6): 1384-96, 2011 Dec 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-22153080
2.
Genetic suppression of transgenic APP rescues Hypersynchronous network activity in a mouse model of Alzeimer's disease.
J Neurosci
; 34(11): 3826-40, 2014 Mar 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-24623762
3.
Amyloid-ß/Fyn-induced synaptic, network, and cognitive impairments depend on tau levels in multiple mouse models of Alzheimer's disease.
J Neurosci
; 31(2): 700-11, 2011 Jan 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-21228179
4.
Delayed postnatal loss of P/Q-type calcium channels recapitulates the absence epilepsy, dyskinesia, and ataxia phenotypes of genomic Cacna1a mutations.
J Neurosci
; 31(11): 4311-26, 2011 Mar 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-21411672
5.
Kv1.1 potassium channel deficiency reveals brain-driven cardiac dysfunction as a candidate mechanism for sudden unexplained death in epilepsy.
J Neurosci
; 30(15): 5167-75, 2010 Apr 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-20392939
6.
Masking epilepsy by combining two epilepsy genes.
Nat Neurosci
; 10(12): 1554-8, 2007 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-17982453
7.
Genetic enhancement of thalamocortical network activity by elevating alpha 1g-mediated low-voltage-activated calcium current induces pure absence epilepsy.
J Neurosci
; 29(6): 1615-25, 2009 Feb 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-19211869
8.
A triplet repeat expansion genetic mouse model of infantile spasms syndrome, Arx(GCG)10+7, with interneuronopathy, spasms in infancy, persistent seizures, and adult cognitive and behavioral impairment.
J Neurosci
; 29(27): 8752-63, 2009 Jul 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-19587282
9.
Deletion of the potassium channel Kv12.2 causes hippocampal hyperexcitability and epilepsy.
Nat Neurosci
; 13(9): 1056-8, 2010 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-20676103
10.
Rai1 deficiency in mice causes learning impairment and motor dysfunction, whereas Rai1 heterozygous mice display minimal behavioral phenotypes.
Hum Mol Genet
; 16(15): 1802-13, 2007 Aug 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-17517686