MUC15 inhibits dimerization of EGFR and PI3K-AKT signaling and is associated with aggressive hepatocellular carcinomas in patients.
Gastroenterology
; 145(6): 1436-48.e1-12, 2013 Dec.
Article
em En
| MEDLINE
| ID: mdl-23933603
Palavras-chave
AKT; EGFR; EMT; ERK; GFP; HCC; HGF; IHC; Liver Cancer; MMP; Mechanism; Mouse Model; OS; PI3K; Prognostic Factor; RTK; TIMP; TTR; epidermal growth factor receptor; epithelial−mesenchymal transition; extracellular regulated kinase; green fluorescent protein; hepatocellular carcinoma; hepatocyte growth factor; immunohistochemistry; mRNA; matrix metallopeptidase; messenger RNA; overall survival; phosphoinositide 3-kinase; receptor tyrosine kinase; time to recurrence; tissue inhibitor of metalloproteinases; v-akt murine thymoma viraloncogene homolog
Texto completo:
1
Coleções:
01-internacional
Base de dados:
MEDLINE
Assunto principal:
Transdução de Sinais
/
Carcinoma Hepatocelular
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Dimerização
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Fosfatidilinositol 3-Quinases
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Proteína Oncogênica v-akt
/
Receptores ErbB
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Neoplasias Hepáticas
/
Mucinas
Tipo de estudo:
Observational_studies
/
Prognostic_studies
/
Risk_factors_studies
Limite:
Animals
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Female
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Humans
Idioma:
En
Ano de publicação:
2013
Tipo de documento:
Article