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SUPT6H controls estrogen receptor activity and cellular differentiation by multiple epigenomic mechanisms.
Bedi, U; Scheel, A H; Hennion, M; Begus-Nahrmann, Y; Rüschoff, J; Johnsen, S A.
Afiliação
  • Bedi U; 1] Center of Experimental Medicine, Department of Tumor Biology, University Medical Center Hamburg-Eppendorf (UKE), Hamburg, Germany [2] Institute of Molecular Oncology, University Medical Center Göttingen, Göttingen, Germany.
  • Scheel AH; 1] Institute of Pathology Nordhessen, Kassel, Germany [2] Center for Pathology and Forensic Medicine, Department of Pathology, University Medical Center Göttingen, Göttingen, Germany.
  • Hennion M; Institute of Molecular Oncology, University Medical Center Göttingen, Göttingen, Germany.
  • Begus-Nahrmann Y; Institute of Molecular Oncology, University Medical Center Göttingen, Göttingen, Germany.
  • Rüschoff J; Institute of Pathology Nordhessen, Kassel, Germany.
  • Johnsen SA; 1] Center of Experimental Medicine, Department of Tumor Biology, University Medical Center Hamburg-Eppendorf (UKE), Hamburg, Germany [2] Institute of Molecular Oncology, University Medical Center Göttingen, Göttingen, Germany.
Oncogene ; 34(4): 465-73, 2015 Jan 22.
Article em En | MEDLINE | ID: mdl-24441044
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fatores de Transcrição / Diferenciação Celular / Receptor alfa de Estrogênio / Epigenômica Tipo de estudo: Prognostic_studies Limite: Female / Humans Idioma: En Ano de publicação: 2015 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fatores de Transcrição / Diferenciação Celular / Receptor alfa de Estrogênio / Epigenômica Tipo de estudo: Prognostic_studies Limite: Female / Humans Idioma: En Ano de publicação: 2015 Tipo de documento: Article