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The chaperone-like protein 14-3-3η interacts with human α-synuclein aggregation intermediates rerouting the amyloidogenic pathway and reducing α-synuclein cellular toxicity.
Plotegher, Nicoletta; Kumar, Dhruv; Tessari, Isabella; Brucale, Marco; Munari, Francesca; Tosatto, Laura; Belluzzi, Elisa; Greggio, Elisa; Bisaglia, Marco; Capaldi, Stefano; Aioanei, Daniel; Mammi, Stefano; Monaco, Hugo L; Samo, Bruno; Bubacco, Luigi.
Afiliação
  • Plotegher N; Department of Biology and.
  • Kumar D; Department of Biochemistry, University of Bologna, Bologna, Italy.
  • Tessari I; Department of Biology and.
  • Brucale M; Department of Biochemistry, University of Bologna, Bologna, Italy, ISMN-National Council of Research, Rome, Italy and.
  • Munari F; Department of Chemical Sciences, University of Padova, Padova, Italy.
  • Tosatto L; Department of Biology and.
  • Belluzzi E; Department of Biology and.
  • Greggio E; Department of Biology and.
  • Bisaglia M; Department of Biology and.
  • Capaldi S; Department of Biotechnology, University of Verona, Verona, Italy.
  • Aioanei D; Department of Biochemistry, University of Bologna, Bologna, Italy.
  • Mammi S; Department of Chemical Sciences, University of Padova, Padova, Italy.
  • Monaco HL; Department of Biotechnology, University of Verona, Verona, Italy.
  • Samo B; Department of Biochemistry, University of Bologna, Bologna, Italy.
  • Bubacco L; Department of Biology and luigi.bubacco@unipd.it.
Hum Mol Genet ; 23(21): 5615-29, 2014 Nov 01.
Article em En | MEDLINE | ID: mdl-24895406
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Proteínas 14-3-3 / Alfa-Sinucleína / Agregação Patológica de Proteínas / Amiloidose Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2014 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Proteínas 14-3-3 / Alfa-Sinucleína / Agregação Patológica de Proteínas / Amiloidose Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2014 Tipo de documento: Article