Plague's partners in crime.

Davis, Kimberly M; Isberg, Ralph R

Davis, Kimberly M; Isberg, Ralph R.

Afiliação

Davis KM; Howard Hughes Medical Institute, Tufts University School of Medicine, 150 Harrison Avenue, Boston, MA 02111, USA; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, 150 Harrison Avenue, Boston, MA 02111, USA.
Isberg RR; Howard Hughes Medical Institute, Tufts University School of Medicine, 150 Harrison Avenue, Boston, MA 02111, USA; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, 150 Harrison Avenue, Boston, MA 02111, USA. Electronic address: ralph.isberg@tufts.edu.

Immunity ; 41(3): 347-349, 2014 Sep 18.

Article em En | MEDLINE | ID: mdl-25238090

ABSTRACT

ABSTRACT

The hallmark of bubonic plague is the presence of grotesquely swollen lymph nodes, called buboes. This frenzied inflammatory response to Yersinia pestis is poorly understood. In this issue of Immunity, St. John et al. (2014) explore the mechanism by which Y. pestis spreads and thus leads to this striking lymphadenopathy.

Assuntos

Linfonodos/patologia; Lisofosfolipídeos/genética; Peste/patologia; Receptores de Lisoesfingolipídeo/imunologia; Esfingosina/análogos & derivados; Yersinia pestis/patogenicidade; Animais; Feminino; Esfingosina/genética

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Peste / Esfingosina / Yersinia pestis / Lisofosfolipídeos / Receptores de Lisoesfingolipídeo / Linfonodos Limite: Animals Idioma: En Ano de publicação: 2014 Tipo de documento: Article

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