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Increases of desmin and α-actinin in mouse cardiac myofibrils as a response to diastolic dysfunction.
Sheng, Juan-Juan; Feng, Han-Zhong; Pinto, Jose R; Wei, Hongguang; Jin, J-P.
Afiliação
  • Sheng JJ; Department of Physiology, Wayne State University School of Medicine, Detroit, MI, USA.
  • Feng HZ; Department of Physiology, Wayne State University School of Medicine, Detroit, MI, USA.
  • Pinto JR; Department of Biomedical Sciences, Florida State University, Tallahassee, FL, USA.
  • Wei H; Department of Physiology, Wayne State University School of Medicine, Detroit, MI, USA.
  • Jin JP; Department of Physiology, Wayne State University School of Medicine, Detroit, MI, USA. Electronic address: jjin@med.wayne.edu.
J Mol Cell Cardiol ; 99: 218-229, 2016 Oct.
Article em En | MEDLINE | ID: mdl-26529187

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Actinina / Disfunção Ventricular / Miócitos Cardíacos / Desmina / Diástole / Miofibrilas Tipo de estudo: Diagnostic_studies / Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Actinina / Disfunção Ventricular / Miócitos Cardíacos / Desmina / Diástole / Miofibrilas Tipo de estudo: Diagnostic_studies / Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article