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P38 pathway as a key downstream signal of connective tissue growth factor to regulate metastatic potential in non-small-cell lung cancer.
Kato, Shinichiro; Yokoyama, Satoru; Hayakawa, Yoshihiro; Li, Luhui; Iwakami, Yusuke; Sakurai, Hiroaki; Saiki, Ikuo.
Afiliação
  • Kato S; Division of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Toyama, Japan.
  • Yokoyama S; Division of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Toyama, Japan. yokoyama@inm.u-toyama.ac.jp.
  • Hayakawa Y; Division of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Toyama, Japan.
  • Li L; Division of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Toyama, Japan.
  • Iwakami Y; Division of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Toyama, Japan.
  • Sakurai H; Department of Cancer Cell Biology, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Toyama, Japan.
  • Saiki I; Division of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Toyama, Japan.
Cancer Sci ; 107(10): 1416-1421, 2016 Oct.
Article em En | MEDLINE | ID: mdl-27403934
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Carcinoma Pulmonar de Células não Pequenas / Sistema de Sinalização das MAP Quinases / Proteínas Quinases p38 Ativadas por Mitógeno / Fator de Crescimento do Tecido Conjuntivo / Neoplasias Pulmonares Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Carcinoma Pulmonar de Células não Pequenas / Sistema de Sinalização das MAP Quinases / Proteínas Quinases p38 Ativadas por Mitógeno / Fator de Crescimento do Tecido Conjuntivo / Neoplasias Pulmonares Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article