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Matrix Metalloproteinase-28 Is a Key Contributor to Emphysema Pathogenesis.
Manicone, Anne M; Gharib, Sina A; Gong, Ke-Qin; Eddy, William E; Long, Matthew E; Frevert, Charles W; Altemeier, William A; Parks, William C; Houghton, A McGarry.
Afiliação
  • Manicone AM; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington. Electronic address: manicone@uw.edu.
  • Gharib SA; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington.
  • Gong KQ; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington.
  • Eddy WE; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington.
  • Long ME; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington.
  • Frevert CW; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington; Department of Comparative Medicine, Center for Lung Biology, University of Washington, Seattle, Washington.
  • Altemeier WA; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington.
  • Parks WC; Women's Guild Lung Institute, Cedars-Sinai Medical Center, Los Angeles, California.
  • Houghton AM; Department of Medicine, Center for Lung Biology, University of Washington, Seattle, Washington; Divisions of Clinical Research and Human Biology, Fred Hutchinson Cancer Research Center, Seattle, Washington.
Am J Pathol ; 187(6): 1288-1300, 2017 Jun.
Article em En | MEDLINE | ID: mdl-28399390

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Enfisema Pulmonar / Metaloproteinases da Matriz Secretadas Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Enfisema Pulmonar / Metaloproteinases da Matriz Secretadas Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article