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FBW7 Loss Promotes Chromosomal Instability and Tumorigenesis via Cyclin E1/CDK2-Mediated Phosphorylation of CENP-A.
Takada, Mamoru; Zhang, Weiguo; Suzuki, Aussie; Kuroda, Taruho S; Yu, Zhouliang; Inuzuka, Hiroyuki; Gao, Daming; Wan, Lixin; Zhuang, Ming; Hu, Lianxin; Zhai, Bo; Fry, Christopher J; Bloom, Kerry; Li, Guohong; Karpen, Gary H; Wei, Wenyi; Zhang, Qing.
Afiliação
  • Takada M; Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, North Carolina.
  • Zhang W; Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory and Department of Molecular and Cell Biology, University of California, Berkeley, California.
  • Suzuki A; Department of Biology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina.
  • Kuroda TS; Open Innovation Center Japan, Bayer Yakuhin, Ltd., Kita-ku, Osaka, Japan.
  • Yu Z; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.
  • Inuzuka H; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
  • Gao D; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
  • Wan L; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
  • Zhuang M; Xin Hua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
  • Hu L; Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, North Carolina.
  • Zhai B; St. Jude Children's Research Hospital, Memphis, Tennessee.
  • Fry CJ; Cell Signaling Technology, Danvers, Massachusetts.
  • Bloom K; Department of Biology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina.
  • Li G; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.
  • Karpen GH; Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory and Department of Molecular and Cell Biology, University of California, Berkeley, California. Qing_Zhang@med.unc.edu ghkarpen@lbl.gov wwei2@bidmc.harvard.edu.
  • Wei W; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts. Qing_Zhang@med.unc.edu ghkarpen@lbl.gov wwei2@bidmc.harvard.edu.
  • Zhang Q; Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, North Carolina. Qing_Zhang@med.unc.edu ghkarpen@lbl.gov wwei2@bidmc.harvard.edu.
Cancer Res ; 77(18): 4881-4893, 2017 09 15.
Article em En | MEDLINE | ID: mdl-28760857
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Autoantígenos / Neoplasias da Mama / Proteínas Cromossômicas não Histona / Transformação Celular Neoplásica / Proteínas Oncogênicas / Neoplasias do Colo / Proteínas de Ciclo Celular / Ciclina E / Instabilidade Cromossômica / Ubiquitina-Proteína Ligases Limite: Female / Humans Idioma: En Ano de publicação: 2017 Tipo de documento: Article
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Autoantígenos / Neoplasias da Mama / Proteínas Cromossômicas não Histona / Transformação Celular Neoplásica / Proteínas Oncogênicas / Neoplasias do Colo / Proteínas de Ciclo Celular / Ciclina E / Instabilidade Cromossômica / Ubiquitina-Proteína Ligases Limite: Female / Humans Idioma: En Ano de publicação: 2017 Tipo de documento: Article