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Angiotensin II Causes ß-Cell Dysfunction Through an ER Stress-Induced Proinflammatory Response.
Chan, Stanley M H; Lau, Yeh-Siang; Miller, Alyson A; Ku, Jacqueline M; Potocnik, Simon; Ye, Ji-Ming; Woodman, Owen L; Herbert, Terence P.
Afiliação
  • Chan SMH; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
  • Lau YS; Department of Pharmacology, Faculty of Medicine, University of Malaya, Kuala Lumpur 50603, Malaysia.
  • Miller AA; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
  • Ku JM; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
  • Potocnik S; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
  • Ye JM; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
  • Woodman OL; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
  • Herbert TP; School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology University, Bundoora, Victoria 3083, Australia.
Endocrinology ; 158(10): 3162-3173, 2017 10 01.
Article em En | MEDLINE | ID: mdl-28938442
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Angiotensina II / Células Secretoras de Insulina / Estresse do Retículo Endoplasmático / Inflamação Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article
Texto completo
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XML
PubMed Links
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Angiotensina II / Células Secretoras de Insulina / Estresse do Retículo Endoplasmático / Inflamação Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article