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Dysfunction of different cellular degradation pathways contributes to specific ß-amyloid42-induced pathologies.
Ji, Xuan-Ru; Cheng, Kuan-Chung; Chen, Yu-Ru; Lin, Tzu-Yu; Cheung, Chun Hei Antonio; Wu, Chia-Lin; Chiang, Hsueh-Cheng.
Afiliação
  • Ji XR; Department of Pharmacology, National Cheng-Kung University, Tainan, Taiwan.
  • Cheng KC; Department of Pharmacology, National Cheng-Kung University, Tainan, Taiwan.
  • Chen YR; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, Taiwan; and.
  • Lin TY; Department of Pharmacology, National Cheng-Kung University, Tainan, Taiwan.
  • Cheung CHA; Department of Pharmacology, National Cheng-Kung University, Tainan, Taiwan.
  • Wu CL; Department of Pharmacology, National Cheng-Kung University, Tainan, Taiwan.
  • Chiang HC; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, Taiwan; and.
FASEB J ; 32(3): 1375-1387, 2018 03.
Article em En | MEDLINE | ID: mdl-29127191
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fragmentos de Peptídeos / Autofagia / Peptídeos beta-Amiloides / Ubiquitina / Complexo de Endopeptidases do Proteassoma / Doença de Alzheimer / Proteólise Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2018 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fragmentos de Peptídeos / Autofagia / Peptídeos beta-Amiloides / Ubiquitina / Complexo de Endopeptidases do Proteassoma / Doença de Alzheimer / Proteólise Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2018 Tipo de documento: Article