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Altered homeostasis and development of regulatory T cell subsets represent an IL-2R-dependent risk for diabetes in NOD mice.
Dwyer, Connor J; Bayer, Allison L; Fotino, Carmen; Yu, Liping; Cabello-Kindelan, Cecilia; Ward, Natasha C; Toomer, Kevin H; Chen, Zhibin; Malek, Thomas R.
Afiliação
  • Dwyer CJ; Department of Microbiology and Immunology, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Bayer AL; Department of Microbiology and Immunology, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Fotino C; Diabetes Research Institute, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Yu L; Diabetes Research Institute, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Cabello-Kindelan C; Barbara Davis Center for Childhood Diabetes, University of Colorado School of Medicine, Aurora, CO 80045, USA.
  • Ward NC; Diabetes Research Institute, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Toomer KH; Department of Microbiology and Immunology, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Chen Z; Department of Microbiology and Immunology, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
  • Malek TR; Department of Microbiology and Immunology, Miller School of Medicine, University of Miami, Miami, FL 33136, USA.
Sci Signal ; 10(510)2017 Dec 19.
Article em En | MEDLINE | ID: mdl-29259102

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Linfócitos T Reguladores / Diabetes Mellitus Experimental / Diabetes Mellitus Tipo 1 / Subunidade beta de Receptor de Interleucina-2 Tipo de estudo: Etiology_studies / Prognostic_studies / Risk_factors_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Linfócitos T Reguladores / Diabetes Mellitus Experimental / Diabetes Mellitus Tipo 1 / Subunidade beta de Receptor de Interleucina-2 Tipo de estudo: Etiology_studies / Prognostic_studies / Risk_factors_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article