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Silibinin-induced endoplasmic reticulum stress and mitochondrial dysfunction suppress growth of endometriotic lesions.
Ham, Jiyeon; Kim, Jonggun; Bazer, Fuller W; Lim, Whasun; Song, Gwonhwa.
Afiliação
  • Ham J; Department of Biotechnology, Institute of Animal Molecular Biotechnology, Korea University, Seoul, Republic of Korea.
  • Kim J; Department of Biotechnology, Institute of Animal Molecular Biotechnology, Korea University, Seoul, Republic of Korea.
  • Bazer FW; Department of Animal Science, Center for Animal Biotechnology and Genomics, Texas A&M University, College Station, Texas.
  • Lim W; Department of Biomedical Sciences, Catholic Kwandong University, Gangneung, Republic of Korea.
  • Song G; Department of Biotechnology, Institute of Animal Molecular Biotechnology, Korea University, Seoul, Republic of Korea.
J Cell Physiol ; 234(4): 4327-4341, 2019 04.
Article em En | MEDLINE | ID: mdl-30144397

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proliferação de Células / Endometriose / Endométrio / Estresse do Retículo Endoplasmático / Silibina / Anti-Inflamatórios / Mitocôndrias / Antioxidantes Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proliferação de Células / Endometriose / Endométrio / Estresse do Retículo Endoplasmático / Silibina / Anti-Inflamatórios / Mitocôndrias / Antioxidantes Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article