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PRMT2ß suppresses autophagy and glycolysis pathway in human breast cancer MCF-7 cell lines.
Chen, Yajun; Dai, Xianpeng; Yao, Yao; Wang, Jing; Yang, Xinzhi; Zhang, Yunsheng; Yang, Jing; Cao, Renxian; Wen, Gebo; Zhong, Jing.
Afiliação
  • Chen Y; Institute of Clinical Medicine, The First Affiliated Hospital of University of South China, Hengyang, China.
  • Dai X; Department of Metabolism and Endocrinology, The Second Affiliated Hospital of University of South China, Hengyang, China.
  • Yao Y; Department of General Surgery, The Second Affiliated Hospital of University of South China, Hengyang, China.
  • Wang J; Institute of Clinical Medicine, The First Affiliated Hospital of University of South China, Hengyang, China.
  • Yang X; Institute of Clinical Medicine, The First Affiliated Hospital of University of South China, Hengyang, China.
  • Zhang Y; Institute of Clinical Medicine, The First Affiliated Hospital of University of South China, Hengyang, China.
  • Yang J; Institute of Clinical Medicine, The Second Affiliated Hospital of University of South China, Hengyang, China.
  • Cao R; Institute of Clinical Medicine, The First Affiliated Hospital of University of South China, Hengyang, China.
  • Wen G; Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, China.
  • Zhong J; Institute of Clinical Medicine, The First Affiliated Hospital of University of South China, Hengyang, China.
Acta Biochim Biophys Sin (Shanghai) ; 51(3): 335-337, 2019 Mar 01.
Article em En | MEDLINE | ID: mdl-30883646
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proteína-Arginina N-Metiltransferases / Autofagia / Peptídeos e Proteínas de Sinalização Intracelular / Glicólise Limite: Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proteína-Arginina N-Metiltransferases / Autofagia / Peptídeos e Proteínas de Sinalização Intracelular / Glicólise Limite: Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article