Hepcidin is not essential for mediating testosterone's effects on erythropoiesis.

Guo, W; Schmidt, P J; Fleming, M D; Bhasin, S

Guo, W; Schmidt, P J; Fleming, M D; Bhasin, S.

Afiliação

Guo W; Research Program in Men's Health: Aging and Metabolism, The Boston Claude D. Pepper Older Americans Independence Center, Brigham and Women's Hospital, and Harvard Medical School, Boston, MA, USA.
Schmidt PJ; Department of Pathology, Boston Children's Hospital and Harvard Medical School, Boston, MA, USA.
Fleming MD; Department of Pathology, Boston Children's Hospital and Harvard Medical School, Boston, MA, USA.
Bhasin S; Research Program in Men's Health: Aging and Metabolism, The Boston Claude D. Pepper Older Americans Independence Center, Brigham and Women's Hospital, and Harvard Medical School, Boston, MA, USA.

Andrology ; 8(1): 82-90, 2020 01.

Article em En | MEDLINE | ID: mdl-31001931

Assuntos

Eritropoese/efeitos dos fármacos; Hepatócitos/metabolismo; Hepcidinas/metabolismo; Receptores Androgênicos/metabolismo; Testosterona/farmacologia; Animais; Feminino; Hepcidinas/genética; Masculino; Camundongos Endogâmicos C57BL; Camundongos Knockout

Palavras-chave

androgen receptor; erythropoiesis; hepcidin; iron; testosterone

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Testosterona / Receptores Androgênicos / Hepatócitos / Eritropoese / Hepcidinas Limite: Animals Idioma: En Ano de publicação: 2020 Tipo de documento: Article

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google