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Erythrocyte ion content and dehydration modulate maximal Gardos channel activity in KCNN4 V282M/+ hereditary xerocytosis red cells.
Rivera, Alicia; Vandorpe, David H; Shmukler, Boris E; Andolfo, Immacolata; Iolascon, Achille; Archer, Natasha M; Shabani, Estela; Auerbach, Michael; Hamerschlak, Nelson; Morton, James; Wohlgemuth, Jay G; Brugnara, Carlo; Snyder, L Michael; Alper, Seth L.
Afiliação
  • Rivera A; Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
  • Vandorpe DH; Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
  • Shmukler BE; Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
  • Andolfo I; Department of Molecular Medicine and Medical Biotechnologies, "Federico II" University of Naples, CEINGE Biotecnologie Avanzate, Naples, Italy.
  • Iolascon A; Department of Molecular Medicine and Medical Biotechnologies, "Federico II" University of Naples, CEINGE Biotecnologie Avanzate, Naples, Italy.
  • Archer NM; Division of Hematology and Oncology, Boston Children's Hospital, Dana-Farber Cancer Center, Department of Pediatrics, Harvard Medical School, Boston, Massachusetts.
  • Shabani E; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, Massachusetts.
  • Auerbach M; Auerbach Hematology-Oncology, Rosedale, Maryland.
  • Hamerschlak N; Department of Hematology, Hospital Israelita Albert Einstein, Sao Paulo, Brazil.
  • Morton J; Quest Diagnostics, San Juan Capistrano, California.
  • Wohlgemuth JG; Quest Diagnostics, San Juan Capistrano, California.
  • Brugnara C; Department of Laboratory Medicine, Boston Children's Hospital and Department of Pathology, Harvard Medical School, Boston, Massachusetts.
  • Snyder LM; Quest Diagnostics, Marlborough, Massachusetts.
  • Alper SL; Departments of Medicine and Laboratory Medicine, University of Massachusetts Medical Center, Worcester, Massachusetts.
Am J Physiol Cell Physiol ; 317(2): C287-C302, 2019 08 01.
Article em En | MEDLINE | ID: mdl-31091145
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Potássio / Hidropisia Fetal / Eritrócitos / Canais de Potássio Ativados por Cálcio de Condutância Intermediária / Anemia Hemolítica Congênita Tipo de estudo: Diagnostic_studies / Observational_studies / Risk_factors_studies Limite: Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Potássio / Hidropisia Fetal / Eritrócitos / Canais de Potássio Ativados por Cálcio de Condutância Intermediária / Anemia Hemolítica Congênita Tipo de estudo: Diagnostic_studies / Observational_studies / Risk_factors_studies Limite: Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article