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RNA-Binding Protein HuR Promotes Th17 Cell Differentiation and Can Be Targeted to Reduce Autoimmune Neuroinflammation.
Chen, Jing; Martindale, Jennifer L; Abdelmohsen, Kotb; Kumar, Gaurav; Fortina, Paolo M; Gorospe, Myriam; Rostami, Abdolmohamad; Yu, Shiguang.
Afiliação
  • Chen J; Department of Neurology, Thomas Jefferson University, Philadelphia, PA 19107; jing.chen@jefferson.edu shiguang.yu@jefferson.edu.
  • Martindale JL; Laboratory of Genetics and Genomics, National Institute on Aging-Intramural Research Program, National Institutes of Health, Baltimore, MD 21224; and.
  • Abdelmohsen K; Laboratory of Genetics and Genomics, National Institute on Aging-Intramural Research Program, National Institutes of Health, Baltimore, MD 21224; and.
  • Kumar G; Department of Cancer Biology, Thomas Jefferson University, Philadelphia, PA 19107.
  • Fortina PM; Department of Cancer Biology, Thomas Jefferson University, Philadelphia, PA 19107.
  • Gorospe M; Laboratory of Genetics and Genomics, National Institute on Aging-Intramural Research Program, National Institutes of Health, Baltimore, MD 21224; and.
  • Rostami A; Department of Neurology, Thomas Jefferson University, Philadelphia, PA 19107.
  • Yu S; Department of Neurology, Thomas Jefferson University, Philadelphia, PA 19107; jing.chen@jefferson.edu shiguang.yu@jefferson.edu.
J Immunol ; 204(8): 2076-2087, 2020 04 15.
Article em En | MEDLINE | ID: mdl-32169842

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Diferenciação Celular / Encefalomielite Autoimune Experimental / Células Th17 / Proteína Semelhante a ELAV 1 / Inflamação Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2020 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Diferenciação Celular / Encefalomielite Autoimune Experimental / Células Th17 / Proteína Semelhante a ELAV 1 / Inflamação Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2020 Tipo de documento: Article