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Sudden Cardiac Arrest in a Patient With Mitral Valve Prolapse and LMNA and SCN5A Mutations.
Mahajan, Asha M; Itan, Yuval; Cerrone, Marina; Horowitz, James; Borneman, Linda; Chinitz, Larry; Jankelson, Lior.
Afiliação
  • Mahajan AM; Leon H. Charney Division of Cardiology, New York University School of Medicine, New York, New York, USA.
  • Itan Y; Department of Genetics and Genomic Sciences, Mount Sinai School of Medicine, New York, New York, USA.
  • Cerrone M; The Charles Bronfman Institute for Personalized Medicine, Mount Sinai School of Medicine, New York, New York, USA.
  • Horowitz J; Leon H. Charney Division of Cardiology, New York University School of Medicine, New York, New York, USA.
  • Borneman L; Leon H. Charney Division of Cardiology, New York University School of Medicine, New York, New York, USA.
  • Chinitz L; Leon H. Charney Division of Cardiology, New York University School of Medicine, New York, New York, USA.
  • Jankelson L; Leon H. Charney Division of Cardiology, New York University School of Medicine, New York, New York, USA.
JACC Case Rep ; 3(2): 242-246, 2021 Feb.
Article em En | MEDLINE | ID: mdl-34317510
Bileaflet mitral valve prolapse (Bi-MVP) is associated with increased risk for cardiac arrest. We describe a patient who presented after a cardiac arrest with Bi-MVP and variants in Lamin A/C (LMNA) and the sodium channel alpha-subunit 5a (SCN5A). Genetic variants may be the culprit for arrhythmogenesis in Bi-MVP patients. (Level of Difficulty: Intermediate.).
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Ano de publicação: 2021 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Ano de publicação: 2021 Tipo de documento: Article